Mathematical Model for the Quantification of Pulmonary and Intrapulmonary Arteriovenous Anastomoses Blood Flow
<p id="p1">Blood flow quantification through intrapulmonary arteriovenous anastomoses (IPAVA) is vital because IPAVA blood flow (Q<sub>IPAVA</sub>) prevents gas exchange and can provide a pathway for emboli to bypass the pulmonary microcirculation and provoke myocardial infarction, transient ischemi...
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| Autor principal: | |
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| Formato: | Capítulo de libro |
| Lenguaje: | inglés |
| Publicado: |
IntechOpen
2024
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| Acceso en línea: | https://www.intechopen.com/chapters/89289 |
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| Sumario: | <p id="p1">Blood flow quantification through intrapulmonary arteriovenous anastomoses (IPAVA) is vital because IPAVA blood flow (Q<sub>IPAVA</sub>) prevents gas exchange and can provide a pathway for emboli to bypass the pulmonary microcirculation and provoke myocardial infarction, transient ischemic attack (TIA), or stroke. Some current techniques allow only an approximate estimate and present technical evaluation difficulties that can compromise the result. To overcome the limitations and obtain an accurate detection of Q<sub>IPAVA</sub>, it is necessary to isolate the fundamental parameters of gas exchange associated with chemical-physical laws that regulate them and develop a specific algebraic model. Therefore, literal equations were elaborated and some experimental data, obtained through noninvasive and easy-to-use methods, were entered. These equations quantified the pulmonary blood flow that permits gas exchange and the one that does not (Q<sub>IPAVA</sub> or generally shunt). Because they are accurate and based on the law of conservation of mass, the use of these equations will allow a general advance in understanding the consequences of Q<sub>IPAVA</sub> on physiological and pathophysiological processes.</p> |
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| ISBN: | 978-0-85014-264-8 |
| DOI: | 10.5772/intechopen.113952 |